CHAPTER 4, PHARMACOTHERAPEUTICS, ENDOCRINE SYSTEM

ENDOCRINE SYSTEM

PREPARED BY MR. ABHIJIT DAS

DIABETES

DEFINITION
Diabetes is a condition of high blood sugar caused by problems with insulin, a hormone that helps glucose enter cells for energy.

  • Type 1: Immune system destroys insulin-producing cells in the pancreas.
  • Type 2: Body becomes resistant to insulin or doesn’t produce enough.

ETIOPATHOGENESIS

  • Type 1: Caused by genetic and environmental factors (e.g., viral infections) leading to immune attacks on insulin cells.
  • Type 2: Caused by insulin resistance and reduced insulin production, often linked to obesity, inactivity, and genetics.
  • Other types:
    • Gestational diabetes: Hormonal changes during pregnancy cause insulin resistance.

CLINICAL MANIFESTATIONS

  • Thirst, frequent urination
  • Weight loss, fatigue, blurred vision
  • Slow-healing wounds, frequent infections
  • Numbness/tingling in extremities
  • Dry, itchy skin, increased hunger
  • Mood changes

NON-PHARMACOLOGICAL MANAGEMENT

1.    Diet: Balanced, high-fiber diet with lean protein; limit sugars and fats.

2.    Exercise: At least 150 minutes/week of moderate activity.

3.    Weight Management: Healthy weight through diet and exercise.

4.    Blood Glucose Monitoring: Regular checks to manage levels.

5.    Stress Management: Techniques like deep breathing or yoga.

6.    Quit Smoking: Reduces risk of complications.

PHARMACOLOGICAL MANAGEMENT

1.    Metformin: Reduces liver glucose production, improves insulin sensitivity.

2.    Insulin: Needed for Type 1 and sometimes Type 2.

3.    Sulfonylureas: Stimulate insulin production (e.g., glipizide).

4.    DPP-4 Inhibitors: Increase GLP-1 to stimulate insulin (e.g., sitagliptin).

5.    GLP-1 Agonists: Mimic GLP-1 to increase insulin and reduce appetite (e.g., exenatide).

6.    SGLT2 Inhibitors: Increase glucose excretion in urine (e.g., canagliflozin).

 

HYPOTHYROIDISM

DEFINITION
Hypothyroidism is a condition where the thyroid gland does not produce enough thyroid hormones.

ETIOPATHOGENESIS

1.    Autoimmune disorders: Hashimoto’s thyroiditis, where the immune system attacks the thyroid.

2.    Thyroid surgery: Removing all or part of the thyroid can cause hypothyroidism.

3.    Radiation therapy: Can damage the thyroid, leading to reduced hormone production.

4.    Medications: Drugs like lithium, interferon, and amiodarone can affect thyroid function.

5.    Congenital hypothyroidism: Some babies are born with an underactive thyroid.

6.    Iodine deficiency: Low iodine intake hinders hormone production.

7.    Pituitary disorders: Issues with TSH production from the pituitary gland can lower thyroid hormones.

CLINICAL MANIFESTATIONS

  • Fatigue, weakness, and weight gain
  • Cold intolerance, dry skin, and hair
  • Constipation, depression, muscle stiffness
  • Joint pain, slow heart rate, and menstrual irregularities

NON-PHARMACOLOGICAL MANAGEMENT

1.    Diet: Include iodine (seaweed, dairy) and selenium (brazil nuts, tuna).

2.    Exercise: Helps reduce fatigue and maintain weight.

3.    Stress management: Yoga, meditation, or breathing exercises.

4.    Supplements: Omega-3 and L-tyrosine may help but require further research.

5.    Avoid certain foods: Limit soy and cruciferous vegetables (e.g., broccoli).

PHARMACOLOGICAL MANAGEMENT

1.    Levothyroxine: Synthetic thyroid hormone, primary treatment.

2.    Combination therapy: Sometimes, levothyroxine and liothyronine (T3) are used together.

 

HYPERTHYROIDISM

DEFINITION
Hyperthyroidism is a condition where the thyroid gland produces too much thyroid hormone.

ETIOPATHOGENESIS

1.    Graves' disease: Autoimmune disorder causing excess hormone production.

2.    Toxic multinodular goiter: Multiple thyroid nodules produce extra hormones.

3.    Thyroiditis: Thyroid inflammation releases stored hormones temporarily.

4.    Excessive iodine: High iodine intake can stimulate overproduction of hormones.

5.    Tumors: Rare tumors in the thyroid or pituitary may cause hormone excess.

6.    Medications: Drugs like amiodarone and lithium can trigger hyperthyroidism.

CLINICAL MANIFESTATIONS

  • Weight loss despite increased appetite
  • Heat intolerance, excessive sweating
  • Palpitations and rapid heartbeat
  • Tremors in hands or fingers
  • Anxiety, irritability, and insomnia
  • Frequent bowel movements, muscle weakness

NON-PHARMACOLOGICAL MANAGEMENT

1.    Diet: Balanced diet; avoid excess iodine, caffeine, and alcohol.

2.    Stress reduction: Practice meditation, deep breathing, or yoga.

3.    Regular exercise: Helps manage anxiety and boosts health.

4.    Cooling techniques: Wear breathable clothes, use fans or AC.

5.    Adequate rest: Prioritize sleep to reduce fatigue.

PHARMACOLOGICAL MANAGEMENT

1.    Antithyroid drugs: Medications like methimazole reduce hormone production.

2.    Beta blockers: Drugs like propranolol manage tremors, palpitations, and anxiety.

3.    Radioactive iodine therapy: Selectively destroys overactive thyroid cells; used if other treatments aren’t effective.

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